Module D- Cardiovascular Certification

The roles of various conditions in cardiovascular disease—such as dysglycemia, insulin resistance and diabetes mellitus— are presented. Methods for the prevention and treatment of cardiovascular disease, including stem cells and chelation therapy, are explored, in addition to occupational risk factors for heart disease, the effects of toxins in the heart and the role of solvents in the development of arrhythmias.

Upon completion of the module, the participant will:

  • Identify the sources of acute and chronic toxic metal exposure (i.e. lead, mercury, cadmium, arsenic)
  • Recognize the adverse health affects of toxic metals on cardiovascular disease and overall health
  • Distinguish patients with symptoms of toxic metal accumulation.
  • Select appropriate tests for diagnosing toxic metal exposure
  • Understand treatment strategies for reducing toxic metal exposure and accumulation; including the use of various chelating agents.
  • Understand the physiological changes to the arteries that increase plaque formation
  • Understand the role of hyperglycemia in the formation of plaque
  • Understand the metabolic continuum of non diabetes, prediabetes and diabetes
  • Be able to discuss ways to improve glycemic control in diabetes
  • Understand AGE and RAGE
  • Understand the role of the mitochondria in the pathophysiology of CVD
  • Understand the PIK3 and MAPK pathways
  • Understand causes of Insulin Resistance
  • Understand how over expression of the MAPK pathway leads to the changes seen in vascular diseases
  • Understand the role of IR in inflammation and oxidation
  • Understand the role of IR in AGE and clotting
  • Be able to come up with clinical ways to decrease the implications of insulin resistance
  • Understand the metabolic syndrome and
  • What causes it
  • How to identify patients before they have the full syndrome
  • What can be done to prevent it
  • What can be done to treat it
  • Understand how a breakdown in glucose homeostasis causes Metabolic syndrome
  • Understand the difference between T1DM and Type2DM
  • Understand how changes in mitochondrial function predispose patients to CVD
  • Understand how increased FFA seen with both increase the risk for CVD
  • Understand how each can increase the risk for HTN and clotting
  • Understand how someone who is insulin sensitive at birth ends up developing T2DM. 
  • Be able to come up with strategies for avoiding or treating T2DM.
  • To create awareness about oral health and systemic concerns with a focus on vascular disease 
  • To introduce new concepts in periodontal diagnosis; a large benefit to dentists and physicians
  • To explore practice advancement and improved patient health through the collaborative relationship
  • To identify cardiovascular drug nutrient depletions
  • To be prepared to recommend the appropriate nutrients and the dosing required for genomic changes and nutrient repletion
  • To be able to identify symptoms associated with drug related nutrient depletions. 
  • To discuss how and why the wheat of 2012 is not the wheat of 1960.
  • To discuss wheat gliadin, amylopectin and lectin.
  • To discuss why elimination of modern wheat can unexpectedly be the most important strategy in improved health and weight loss.
  • Discuss how grains, such as wheat, corn, rye, millet, and others, are recent additions to the diet of Homo sapiens, departures from the diet that humans have consumed for most of their evolutionary history.
  • Review the evidence that, when wild grains were first incorporated into the human diet, adverse health effects results, especially those involving the teeth and bones, the evidence that dominates the archaeological evidence.
  • Discuss how grains are responsible for a collection of metabolic distortions that constitute a situation that leads to coronary artery disease: Grain consumption is a coronary risk factor.